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- Current Students
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Student Resources
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Student Publications
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Bae, JuYun
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Boehm, Bayli
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Boley, Patricia
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Bolterstein, Elyse
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Booth, Clarissa
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Brody, Matthew
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Bultman, JoAnna
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Desotelle, Josh
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Elmergreen, Tammy
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Hutchinson, John
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Irving, Amy
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Irving, Roy
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Johnson, Brian
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Johnson, Delinda
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Jung-Hynes, Brittney
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Kumar, Kartik
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Lee, Narae
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Lorch, Jeff
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Mehta, Vatsal
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Novick, Rachel
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Park, Heesoo
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Pham, Ly
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Rhoads, Keelia
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Rufer, Echoleah
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Sand, Jordan
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Schmit, Travis
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Shan, Weihua
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Shanle, Erin
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Shetty, Ameesha
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Syed, Deeba
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Tarapore, Rohinton
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Wiecinski, Paige
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Wong, Letitia
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Yang, Sarah
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Yu, Min
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Zhao, Yun
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Calkins, Marcus
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Roberts, J. Lea
J. Lea Roberts
Masters Candidate - Started 2004
Native of Minnesota
Lab of David Jarrard, PhD
Contact Information
Email: J. Lea Roberts
Undergraduate Work
University of wisconsin-Superior
Bachelor of Science, Biology/Chemistry (2002)
Research as of Spring 2005
My research will focus on the effects of aging, environmental toxins, and oxidative stress on the incidence of prostate cancer. Ongoing projects I have joined look at two aims for understanding possible mechanisms of cancer development. Aim 1 hypothesizes a change in imprinting of the prostate from aging. Aim 2 asks if imprinting is modulated by methylation. A change in genomic imprinting may be a factor in increased susceptibility to cancer. I will test tissues collected from animals maintained by the lab to answer these questions.
Since joining Dr. Jarrard’s lab, we have been developing a new project to look at the effects of prenatal TCDD exposure on reproductive system development of male mice. Changes in imprinting, methylation, and function will be examined. This project is a collaborative effort with the lab of Dr. Richard Peterson. I will take the lead for collection and testing of tissues, as well as compilation of the results. This project is expected to last approximately one year and result in publication.
Future plans include the development of additional toxin exposure models and microarray analysis of genes exposed to a variety of potentially dangerous chemicals. Investigation into a potential toxicological link to the rise in prostate cancer incidence will be included. The incidence increases as a population ages, but the current rate of increase cannot be accounted for by aging alone.
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