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Bae, JuYun
Boehm, Bayli
Boley, Patricia
Bolterstein, Elyse
Brody, Matthew
Bultman, JoAnna
Calkins, Marcus
Desotelle, Josh
Dever, Joe
Feld, Mara
Hutchinson, John
Irving, Roy
Johnson, Brian
Johnson, Delinda
Jung, Brittney
Klingbiel, Sarah
Mehta, Vatsal
Novick, Rachel
Pham, Ly
Rhoads, Keelia
Rufer, Echoleah
Sand, Jordan
Schmit, Travis
Shan, Weihua
Shetty, Ameesha
Tarapore, Rohinton
Wiecinski, Paige
Yu, Min
Hutchinson, John

John Hutchinson - Email
PhD Candidate - Started 2007
Sparta, WI
Lab of Randall Tibbetts, PhD

Undergraduate Work
University of Wisconsin-Madison (2002-2006)
Bachelors of Science-Biology (2006)

Research as of Spring 2008
Ataxia telagniectasia (A-T) is a rare, inherited disease characterized by radiation sensitivity, immune deficiency, chromosome instability, predisposition to cancer, and progressive cerebellar degeneration. A-T is caused by mutations in the ATM (ataxia-telangiectasia-mutated) gene. ATM encodes a protein kinase involved in DNA repair and cell cycle checkpoint signaling in response to DNA damage. The focus of my research is to understand the mechanisms of cerebellar degeneration in A-T and provide new insights into ATM function and regulation.

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Date Last Updated: 07/21/2008 webteam@med.wisc.edu